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PreviewIssue DateTitleAuthor(s)
2014Treatment with the NK1 antagonist emend reduces blood brain barrier dysfunction and edema formation in an experimental model of brain tumorsHarford-Wright, E.; Lewis, K.; Ghabriel, M.; Vink, R.; Alonso, M.
2014Advancing drug therapy for brain tumours: a current review of the pro-inflammatory peptide substance P and its antagonists as anti-cancer agentsMander, K.; Harford-Wright, E.; Lewis, K.; Vink, R.
2014Evaluating the role of substance P in the growth of brain tumorsHarford-Wright, E.; Lewis, K.; Vink, R.; Ghabriel, M.N.
2013Characterisation of Walker 256 breast carinoma cells from two tumour cell banks as assessed using two models of secondary brain tumoursLewis, K.; Harford-Wright, E.; Vink, R.; Ghabriel, M.
2013Walker 256 tumour cells increase substance P immunoreactivity locally and modify the properties of the blood-brain barrier during extravasation and brain invasionLewis, K.; Harford-Wright, E.; Vink, R.; Nimmo, A.; Ghabriel, M.
2013NK1 receptor antagonists and dexamethasone as anticancer agents in vitro and in a model of brain tumours secondary to breast cancerLewis, K.; Harford-Wright, E.; Vink, R.; Ghabriel, M.
2012Targeting classical but not neurogenic inflammation reduces peritumoral oedema in secondary brain tumoursLewis, K.; Harford-Wright, E.; Vink, R.; Ghabriel, M.
2011Towards drug discovery for brain tumours: interaction of kinins and tumours at the blood brain barrier interfaceHarford-Wright, E.; Lewis, K.; Vink, R.
2013The potential for substance P antagonists as anti-cancer agents in brain tumoursHarford-Wright, E.; Lewis, K.; Vink, R.

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